RT Book, Section
A1 Thomas Thach, W.
A2 Watts, Ray L.
A2 Standaert, David G.
A2 Obeso, Jose A.
SR Print(0)
ID 55803130
T1 Chapter 41. Physiology and Pathophysiology of the Cerebellum, and Rehabilitation Strategies after Cerebellar Injury
T2 Movement Disorders, 3e
YR 2012
FD 2012
PB The McGraw-Hill Companies
PP New York, NY
SN 978-0-07-161312-5
LK accessphysiotherapy.mhmedical.com/content.aspx?aid=55803130
RD 2024/04/18
AB At  the  turn  of  the  last  century,  Flourens  and  Luciani  in  animals  and  Babinski  and  Holmes  in  humans  described  behavioral  deficits  caused  by  focal  cerebellar  lesions,  and  they  speculated  about  the  causal  mechanism.  Perhaps  most  influential  were  the  studies  of  Gordon  Holmes,  who  meticulously  documented  the  movement  deficits  associated  with  cerebellar  gunshot  wounds  in  World  War  I.1,2  He  insisted  that  these  deficits  may  be  attributed  to  cerebellar  disease  only  in  patients  who  have  normal  strength  and  somesthesis.  Yet  even  he  accepted  that  weakness/“asthenia”  may  sometimes  occur  transiently  and  acutely.